What is the cause of most cancers?

The current belief in medical research holds that most cases of breast cancer are caused by a mutation in a certain gene. Genes, in general, produce proteins, which are the building blocks of cells. The concentration of proteins is tightly regulated. A mutated or injured gene produces an abnormal concentration of its protein, which may lead to disease. Take breast cancer as an example. In 1994, Mark Skolnick, PhD, discovered the BRCA1 gene (BRCA1 is short for BReast CAncer 1). Following the discovery, scientists observed a low level of the BRCA1 protein in breast cancer tissues. This observation created a lot of excitement. At the time, scientists believed that they were on the verge of finding the cause of breast cancer. The reasoning was that breast cancer patients must have a mutated BRCA1 gene, which would explain the decreased production of the protein, and the development of tumors.

In the United States, 180,000 cases of breast cancer are diagnosed each year. However, only 5% of these cases have a mutated BRCA1 gene. In more than 95% of the breast cancer patients the gene is not mutated.

So here is the mystery. If the gene is not mutated in the great majority of the breast cancer patients, why are the tumors showing a low level of the BRCA1 protein? The BRCA1 gene is not unique. Many normal (perfect shape, non-mutated) genes exhibit a mysterious abnormal (increased or decreased) production of proteins in cancer.

And breast cancer is only one example. Prostate cancer is another.  For instance, according to Professor Matikaine and colleagues, inherited genetic defects might be the cause of only 5-10% of all prostate cancer cases, [1] and overall the Spanish National Cancer Centre (CNIO) concludes on their website that "A small proportion, that is estimated between 5% and 10% of all tumours, has a hereditary character. Hereditary cancers are the consequence of germinal mutations in specific genes that increase the susceptibility to develop a neoplasia." [2] These observations prompted Dr. Raxit J. Jariwalla to conclude in his European Journal of Cancer paper that "genetic alteration is not the initiating event of cancer." [3]

So, if a genetic alteration (also called genetic mutation or genetic injury) is not the initiating event of most cancers, what is the initiating event?

According to Dr. Hanan Polansky highly acclaimed scientific discovery [4], the cause of most cancers is latent viral DNA that lays hidden in human cells. To survive, the latent viral DNA feeds off the resources it "steals" from the human DNA in these cells, thereby creating a supply shortage for the human genes. The latent viral DNA, which is a genetic parasite, in fact "starves" the human genes, and causes them to behave irregularly, which leads to disease. This effect of the foreign DNA on human genes inspired Dr. Hanan Polansky to call the discovery the "starved" gene phenomenon.

In the case of the BRCA1 gene, the latent viral DNA starves the BRCA1 gene, and prevents it from producing the BRCA1 protein. This is the reason why studies observed a decrease in the concentration of the BRCA1 protein and not mutations in the BRCA1 gene. Moreover, recently several studies report finding viral DNA in large portion of breast cancer tissues, confirming the role of a latent viruses in the development of breast cancer.

Breast cancer is not unique. Similar observations have been reported in other types of cancers.

How do you treat latent viruses?

To maintain a normal rate of cell replication one needs to control the concentration of latent viral DNA in his system. Gene-Eden-VIR, a dietary supplement, can assist in maintaining a healthy level of latent viruses in your body.

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References:

1. Matikaine MP, Pukkala E, Schleutker J, Tammela TL, Koivisto P, Sankila R, Kallioniemi OP. Relatives of prostate cancer patients have an increased risk of prostate and stomach cancers: a population-based, cancer registry study in Finland. Cancer Causes Control. 2001 Apr;12(3):223-30.

2. http://www.cnio.es/ing/programas/prog504a1.asp

3. Jariwalla RJ. Microcompetition and the origin of cancer. Eur J Cancer. 2005 Jan;41(1):15-9

4. Polansky H. Microcompetition with foreign DNA and the origin of chronic disease. CBCD publishing. 2003. 543 p.